By Marshall M. Haith, Janette B. Benson
Infancy is a special interval, in that at no different country is there better development and improvement. in this time, development is going on at a actual point, yet both remarkable are the socio-emotional and cognitive advancements in this time. Genetics, the womb surroundings, and the actual atmosphere after start all mix to affect the speed and demeanour of development. Who we finally develop into as members starts here.
Intended for college and public libraries, the Encyclopedia of boy or girl and Early youth Development is the main reference paintings that offers a accomplished access aspect into the entire latest literature on baby improvement from the fields of psychology, genetics, neuroscience, and sociology. The scope of this paintings is to appreciate the developmental alterations, once they happen, why they ensue, how they ensue, and people elements that impact that improvement. even supposing a few clinical info is integrated, the emphasis is on basic development and is essentially from a mental perspective.
- Cutting aspect content material will surround the time period from neo-nates to age three
- Award-winning Editors-in-Chief to make sure authoritative quality
- Organized alphabetically through subject for ease of reference
- Each article will contain a thesaurus defining any distinct terms
- Suggested readings on the finish of every article supply a source for extra information
- Extensively cross-referenced in the paintings to allow reader entry to complete and suitable details on the topic of their interests
- Online availability through ScienceDirect platform
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Additional info for Psychology Encyclopedia Of Infant And Early Childhood Development
In turn, because she has two X chromosomes, a woman with an X-linked dominant condition has only a 50% chance of transmitting her mutant X chromosome. Thus, half of her sons and half of her daughters, on average, are expected to be affected. As an example of this pattern of transmission, consider Aicardi syndrome, named for the French 23 neurologist who systematically described the syndrome in the 1960s. This syndrome is lethal for males and thus is observed only in females. Its frequency is very low and is estimated at 300–500 cases worldwide.
For example, researchers have shown that the administration of losartan, an AT1 antagonist, to a mouse model of MFS prevents aortic aneurysm in this mouse. This basic work has direct implications for clinical outcomes, creating new hope of reversing cardiac symptomatology for MFS patients. Thus, the initial identification of the FBN1 gene as the basis for the majority of cases of MFS (a genetics finding) leads to understanding the pathways in which this gene is involved and identification of other ‘players’ in this pathway (a genomics finding), which, in turn, leads to understanding proteins interactive within these pathways (a proteomics finding).
Carriers of such a variant of the FMR1 gene do not demonstrate typical fragile X features or behaviors. However, they are at risk for the development of a disorder called fragile X associated tremor/ataxia (FXTAS). FXTAS is a progressive disorder of movement (ataxia), tremors, memory loss, reduced sensation in the lower extremities, and mental and behavioral changes. Women who carry 35–200 copies of the repeat are also at risk for premature ovarian failure, which can result in infertility. The repeat is inherited with an X-linked dominant pattern.